Hemolytic Disease…Erythroblastosis Fetalis
Erythroblastosis Fetalis also known Hemolytic disease of the new born or immune Hydrops Fetalis is a disease in the fetus or new born caused by trans-placental transmission of maternal and fetal blood group incompatibility .
Rh incompatibility may develop when a
woman with Rh negative blood conceives a fetus with Rh Positive blood by a man
with Rh positive blood.
RBC from the fetus leaks across the
placenta and enter the woman's circulation throughout pregnancy with greatest
transfer occurring at delivery. This transfer stimulates maternal antibody
production against the Rh Factor, which is called isoimmunization .In
succeeding pregnancies, the antibodies reach the fetus via placenta and destroy
(lyse) the fetal RBC's .The resulting anemia may be so profound that the fetus
may die in utero. Reacting to the anemia, the fetal bone marrow may release
immature RBC's or erythroblasts into the fetal peripheral circulation causing
Erythroblastosis Fetalis . Maternal - fetal incompatibilities of ABO blood
types leading to Neonatal Erythroblastosis are less severe and less common than
those of the Rh Factor.
The antibodies cause the baby's red
blood cells to be destroyed and baby develops anemia .The baby's body tries to
compensate for anemia by releasing immature red blood cells. The over
production of erythroblasts can cause the liver and spleen to become enlarge
potentially causing liver damage or ruptured spleen .Since the blood lacks
clotting factors, excessive bleeding can be complication .
Babies who survive pregnancy may
develop Kernicterus, leading to deafness speech problems,cerebral palsy or
mental retardation. Extended Hydrops Fetalis can inhibit lung growth and
contribute to heart failure. These serious complications are life threatening
but with good medical treatment the fatality rate is very low.
Written by
Kausar Khan
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